Identifiability of restricted latent class models with binary responses
二元响应受限潜在类别模型的可识别性AI 解读 ↓
本页整理公开书目信息,不生成学术质量评分。AI 解读根据题名及所注明的摘要或原文片段生成,未完成全文核验,可能有误;具体假设、证明与结论请核对原文。摘要保留来源文本,未经模型改写。
DOI 首次登记不等于接收或发表日期。部分更正、评论等条目没有独立摘要;缺少可靠材料时保留待补状态。
引用量采用 OpenAlex 单条关联记录,不合计多个记录;关联记录可能包含正式发表版本,不等于此版本单独引用。缺失不视为零,引用指标不代表论文质量。
关注作者按书目中的完整姓名匹配,同名和姓名变体需人工辨别。相关论文先按共享细分方向和具体方法排列,再参考通用方法与发表日期。
AI 根据所提供的论文材料归类:二元响应潜在类别模型用于社会心理测评,摘要未将响应解释为经济选择或明确提出经济结构估计。
反馈分类问题二元响应受限潜在类别模型的可识别性AI 解读 ↓
正在核对关联版本…
正在核对对应版本的资源…
Statistical latent class models are widely used in social and psychological researches, yet it is often difficult to establish the identifiability of the model parameters. In this paper, we consider the identifiability issue of a family of restricted latent class models, where the restriction structures are needed to reflect pre-specified assumptions on the related assessment. We establish the identifiability results in the strict sense and specify which types of restriction structure would give the identifiability of the model parameters. The results not only guarantee the validity of many of the popularly used models, but also provide a guideline for the related experimental design, where in the current applications the design is usually experience based and identifiability is not guaranteed. Theoretically, we develop a new technique to establish the identifiability result, which may be extended to other restricted latent class models.
Mendelian randomization (MR) infers causal effects of exposures on outcomes using genetic variants as instrumental variables, but existing methods remain vulnerable to bias and loss of power when instruments are invalid. We hypothesize that closely related outcome traits share a large overlap in their valid instruments for a given exposure, so that borrowing instruments across such traits can yield more robust MR inference. To operationalize this, we introduce a novel…
This article develops an identification framework for heterogeneous policy effects when individual-level data are available only in the treated jurisdiction. The framework combines outcome changes from repeated cross-sections within that jurisdiction with a population-average effect identified from aggregate panel data. Under conditional relative parallel trends and overlap, this information identifies subgroup effects standardized to the treated jurisdiction's overall…
Normality pre-tests on reduced-form innovations cannot detect whether two or more underlying structural shocks are Gaussian, and hence cannot detect a failure of identification in non-Gaussian SVARs. We propose a bootstrap-based approach that reveals such a failure. Under the null of valid identification and certain regularity conditions, the conditional bootstrap distribution of the maximum likelihood estimator of the impact matrix is asymptotically normal, so the…
Principal stratification (PS) is a commonly used approach for understanding the mechanisms through which a treatment affects an outcome. The goal of this work is to extend the PS framework to studies with continuous treatments, which introduces a number of both challenges and opportunities in terms of defining causal effects and performing inference. This manuscript provides multiple key methodological contributions: 1) we introduce principal causal estimands for continuous…
度量统计视角下的随机集
Since the seminal work by Beresteanu and Molinari(2008), the random set theory and related inference methods have been widely applied in partially identified econometric models. Meanwhile, there is an emerging field in statistics for studying random objects in metric spaces, called metric statistics. This paper clarifies a relationship between two fundamental concepts in these literatures, the Aumann and Fréchet means, and presents some applications of metric statistics to…
Background: Generalized Odds (Gen Odds) provide summaries for ordinal outcomes without requiring the proportional odds assumption. However, conventional Gen Odds compares independently drawn treatment and control outcomes and targets a population-level contrast rather than an individual-level contrast. We developed an individual-level causal formulation of Gen Odds and examined its identification. Methods: We defined the Causal Gen Odds using the joint distribution of…